Synapses & Drugs
 

Pre- & postsynapse

I. presynapse:
  1. Neurotransmitter: chemicals that influence neighboring neurons
    A. stored in synaptic vesicles
    B. released from presynaptic neuron into synapse when APs occur
 

II. the postsynapse:
  1. neurotransmitter (NT) binds to a receptor
     A. like a key fits into a lock
     B. two types of receptors
        a. ionotropic:
        b. metabotropic:
  2. receptor binding causes postsynaptic potentials
     A. excitatory postsynaptic potentials (EPSPs) graded depolarizations
     B. inhibitory postsynaptic potentials (IPSPs) graded hyperpolarizations
  3. postsynaptic potential are capable of
     A. temporal summation
     B. spatial summation
  4. examples of how postsynaptic potentials occur?
     A. receptor binding causes Na+ ions to enter (EPSPs)
     B. receptor binding causes K+ ions to exit (IPSPs)
  5. What determines if a neurotransmitter (NT) produces IPSPs or EPSPs
     A. the type of neurotransmitter
     B. the type of  postsynaptic receptor
       a. many NT can be either excitatory or inhibitory
 

III. regulation of the neurotransmitter signal
  1. enzymatic deactivation: enzymes breakdown the NT
  2. reuptake: NT is taken back into the presynaptic neuron
  3. autoreceptors: receptors found on the presynaptic neuron
         a. inhibit the synthesis & release of NT
  4. axoaxonic synapse: a synapse between two axons
         a. alter the amount of NT released by the terminal button
 
 

Psychopharmacology

I. drugs can facilitate or oppose neurotransmitter functioning
   1. agonist:
   2. antagonist:
 

II. types of  neurotransmitters (NT) at least 50 have been reported

  1. acetylcholine: main NT of the autonomic nervous system
    A. precursor is choline: from diet
    B. two types of receptors
        a. nicotinic receptors:
         --e.g., of agonist is nicotine
         --e.g., of antagonist is curare
        b. muscarinic receptors:
         --e.g., of agonist is muscarine
         --e.g., of antagonist is atropine (belladonna)
    C. acetylcholinesterase (AChE) deactivates it
    D. botulinum toxin: prevents release from terminal buttons
    E. black widow spider venom: causes terminals to release NT

  2.  monoamines: monoamine oxidase (MAO) deactivates these.
    A. indolamines which include
        a. serotonin (5-HT):
          -precursor is the amino acid tryptophan
          -at lease 7 different receptors (most inhibitory)
          -Prozac is a reuptake inhibitor
          -LSD stimulates at least one type of 5-HT receptor

    B. catecholamines:
        a. norepinephrine (NE) or noradrenalin:
          -precursor is the amino acid tyrosine
          -four different types of receptors

        b. dopamine (DA):
           -precursor is tyrosine
           -at least 5 different DA receptors (most inhibitory)
           -Parkinson’s disease (L-DOPA for treatment)

        c. drugs that influence catecholamines
           -cocaine prevent reuptake
           -amphetamine increases release
           -methylphenidate

   3. amino acid transmitters:
     A. gamma aminobutyric acid (GABA)
        a. precursor is glutamate
        b. two GABA receptors (both inhibitory)
        c. Valium is a GABA agonist
        d. GABA receptors have binding sites for at least 3 different chemicals
           -primary binding site is for GABA
           -a second site binds benzodiazepines (e.g., Valium)
           -a third site binds barbiturates & alcohol
 

     B. glutamic acid (glutamate)
        a. principle excitatory NT in the brain
        b. 5 types of receptors have been identified

     C. glycine
        a. inhibitory NT found in the brainstem & spinal cord

   4. peptide neurotransmitters:
     A. endorphins or enkephalins (endogenous opiates)
        a. these resemble opiates in structure
        b. three types of receptors have been found
        c. opiates (e.g., morphine) are endorphin agonists

  5. purines
     A. adenosine: NT that apparently has a calming effect
       a. caffeine blocks adenosine receptors

  6. anandamide: endogenous THC substance
     A. THC is an agonist (mimics anandamide)

Back to Brain & Human Nature Outlines

Back to Home Page